“In N2a cells, cilostazol (10–30 μM), significantly increased the expression of P-AMPKα ( Thr 172) and downstream P-ACC (acetyl-CoA carboxylase) (Ser 79) as did resveratrol (SIRT1 activator), or AICAR (AMPK activator), which were blocked by KT5720, compound C (AMPK inhibitor), or sirtinol.”