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Last Updated: 8 months ago

Possible Interaction: Potassium and Aspartate



Research Papers that Mention the Interaction

Perfusion with artificial extracellular fluid containing 45 mM potassium produced a significant increase in aspartate and glutamate efflux.
Pharmacological research  •  2006  |  View Paper
Stimulation of the nucleus tractus solitarii with 35 mM potassium in vitro significantly increased extracellular concentrations of aspartate and glutamate.
Neuroscience  •  1996  |  View Paper
Depolarization induced by the high potassium medium (40 mmol/L) enhanced the release of glutamate and aspartate from superfused rat striatum, whereas the efflux of glutamine remained unchanged.
Biomedical chromatography : BMC  •  1989  |  View Paper
In cerebellar granule cells, exposure to potassium resulted in a large (sixfold) and prompt release of D‐[3H]aspartate , largely calcium‐dependent.
Journal of neurochemistry  •  1989  |  View Paper
Sodium ([Na+]o greater … [Na+]i gradients, and potassium ([K+]o less than [K+]i) or proton ([H+]o less … [H+]i) gradients can independently energize the Na+-dependent accumulation of L-aspartate above equilibrium levels, suggesting that Na+ and L-… cotransport occurs with concomitant K+ or H+ antiport.
The American journal of physiology  •  1987  |  View Paper
In both CN and CX, potassium (5-55 mM) produced a concentration-dependent increase in the release of Asp , Glu, Gly, and GABA in the presence of extracellular Ca2+.
Peptides  •  1986  |  View Paper
The uptake of d‐[3H]‐aspartate was found to be stimulated at high potassium in both glutamatergic neurons (98%) and in astrocytes (70%).
Journal of neurochemistry  •  1985  |  View Paper
In the presence of aspartate in higher concentrations the absorption of both magnesium and potassium is inhibited to a certain degree.
Arzneimittel-Forschung  •  1978  |  View Paper
Glutamate, aspartate and high potassium concentrations increase the rate of loss of potassium.
Experimental eye research  •  1972  |  View Paper
Aspartate uptake at pH 8 in the absence of glucose is markedly greater in potassium than in sodium phosphate buffers; the effect of K+ is smaller in the presence of glucose and KCl is less effective than potassium phosphate.
Biochimica et biophysica acta  •  1972  |  View Paper