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“In the presence of Intralipid , the LPL hydrolytic inhibitor tetrahydrolipstatin blocked excess glucose retention in cellular TG but did not significantly decrease glucose disappearance in C2/LPL cells.”
American journal of physiology. Endocrinology and metabolism • 2010 | View Paper
“They show that inhibiting intestinal fat digestion with orlistat increased intragastric self-administration of 30% intralipid (IL30), indicating that digestion of the infused IL30 is necessary for its full satiating potential.”