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Journal of Pharmacology and Experimental Therapeutics • 2008 | View Paper
“ 5-HT release is modulated by a variety of neuromodulators acting on adrenoreceptors, nicotinic and muscarinic cholinoreceptors and 5-HT3 receptors (Racke et al. 1996).”
Clinica chimica acta; international journal of clinical chemistry • 1970 | View Paper
“Exogenous ligands such as galanthamine and the neurotransmitter 5-hydroxytryptamine , when applied in submicromolar concentrations with nicotinic agonists, significantly increase the frequency of opening of nicotinic receptor channels and potentiate agonist-activated currents.”
“S-zacopride, like renzapride and 5-HT , presynaptically inhibited nicotinic fast excitatory postsynaptic potentials, an effect that can be mediated by 5-HT1P or 5-HT1A receptors.”
The American journal of physiology • 1991 | View Paper
“It is, therefore, suggested that diphasic effect of 5-HT on the nicotinic transmission is due mainly to a modulation of the ACh-release from presynaptic nerve terminals.”
Journal of the autonomic nervous system • 1989 | View Paper
“Thus serotonin , probably acting on 5-HT1A receptors, blocks effectively but indiscriminately all cholinergic facilitations, whether mediated by nicotinic or muscarinic receptors.”
Canadian journal of physiology and pharmacology • 1989 | View Paper
“ Fast nicotinic excitatory postsynaptic potentials (EPSPs) and slow EPSPs recorded from S neurons in the myenteric plexus were suppressed by 5-HT , 5-CT, and DPAT; slow EPSPs in myenteric AH neurons were also inhibited by these agonists.”
The American journal of physiology • 1988 | View Paper
“The evoked nicotinic excitatory postsynaptic potential and the adrenergic inhibitory postsynaptic potential were decreased by 5-hydroxytryptamine ; a portion of this inhibition showed desensitization and was blocked by ICS 205-930 as well as by the muscarinic receptor antagonists, atropine and pirenzepine.”