“Lastly, MLT treatment significantly decreased the levels of toll-like receptors 4 (TLR4), phosphorylated-protein kinase B (Akt), and phosphorylated-mechanistic target of rapamycin (mTOR), indicating that blocking TLR4/Akt/mTOR pathway might be an underlying basis for the anti-inflammatory and anti-apoptosis effects of MLT.”
“Lastly, MLT treatment significantly decreased the levels of toll‐like receptors 4 (TLR4), phosphorylated‐protein kinase B (Akt), and phosphorylated‐mechanistic target of rapamycin (mTOR), indicating that blocking TLR4/Akt/mTOR pathway might be an underlying basis for the anti‐inflammatory and anti‐apoptosis effects of MLT.”