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“Neurological examination was normal and there was no clinical evidence of adrenal failure, but the dosage of VLCFAs revealed a marked increase of C26:0 (3.40 μmol/L, n.v.”
“Deleterious activity of VLCFAs on energy-dependent mitochondrial functions declines with increasing the hydrocarbon chain length (C22:0 > C24:0 > C26:0).”