Direct vasodilator effects of physiological hyperinsulin‐aemia in human skeletal muscle

 infusion increased forearm lactate release and 

Effect of insulin on renal sodium and uric acid handling in essential hypertension.

 decreases the urinary excretion of sodium, 

Intravenous infusion or nebulization of salbutamol for treatment of hyperkalemia in patients with chronic renal failure.

 was significantly correlated with the elevation of 

Antinatriuretic action of insulin is preserved after angiotensin I converting enzyme inhibition in normal man.

 infusion reduced both the absolute and fractional urinary excretion rates of sodium (P < 0.001) and 

 (P < 0.001); these effects of insulin were not altered after converting enzyme inhibition.

Glucose modulation of the disposal of an acute potassium load in patients with end-stage renal disease.

 is known to stimulate this disposal by enhancing 

COMPARATIVE STUDY OF DIFFERENT INSULIN REGIMENS IN MANAGEMENT OF DIABETIC KETOACIDOSIS

Block of Kv1.7 potassium currents increases glucose-stimulated insulin secretion

Cyclic activation of voltage‐gated potassium channels (Kv) generates an outward, ‘delayed rectifier’

 current, which drives the repolarizing phase of each spike and modulates 

The alpha2L111R,N122D isoform of the Na,K-ATPase expressed in HeLa cells does not undergo an adipocyte-like increase in activity in response to insulin.

 uptake by a preferential activation of the alpha2 isoform of the Na,K-ATPase.

The role of adrenal medullary catecholamines in potassium homoeostasis.

 in muscle, liver and adipose tissue [2-41.

Serum Electrolyte Levels and Ventricular Arrhythmia

Angiotensin-converting enzyme inhibitors, angiotensin receptor blockers, and

Renal tubular dysfunction in patients with molecular defects of the insulin receptor gene

 and IGFI can activate PI3K and therefore may inhibit 

 excretion by reducing the abundance of ROMK.

Practolol inhibition of some salbutamol-induced metabolic and hormonal responses

 were only partially inhibited, and the possible involvement of the β2-receptors as well in these 2 cases cannot be ruled out.

Effect of oral glucose administration on serum potassium concentration in hemodialysis patients.

 uptake into cells in hemodialysis (HD) patients and healthy subjects.

Early insulin release and its response to potassium supplementation in protein-calorie malnutrition

This suggests that impaired insulin secretion in PCM is in part due to a

Acute postoperative metabolic complications of diabetes.

Its main treatment is an active rehydration and

Purification and Properties of Insulin-Activated Nitric Oxide Synthase from Human Erythrocyte Membranes

Line weaver Burk plot of the purified enzyme showed that the stimulation of the enzymic activity by

Medical and dialytic management of hyperkalemia in hemodialysis patients

 removal, one might anticipate substantial rebound hyperkalemia within a few hours of ending the dialysis session, although this possibility has not been formally evaluated.

Hypokalemia after pediatric albuterol overdose: a case series.

 may also contribute to the intracellular shift of 

Potassium as a link between insulin and the renin-angiotensin-aldosterone system.

 also appears to play a key role in the antinatriuretic effect of 

Independent stimulation of glucose metabolism and Na+-K+ exchange by insulin in the human forearm.

 uptake into skeletal muscle by stimulating the activity of the Na+-K+ pump.

Insulin-mediated hypokalemia and paralysis in familial hypokalemic periodic paralysis.

 (and other factors) may be a central feature of this syndrome.

Potassium metabolism.

 and aldosterone secretion and increases Na-K ATPase in the distal nephron, so promoting its own redistribution or excretion.

Insulin: fundamental mechanism of action and the heart.

 hyperpolarizes the cell, a change in the ratio of intracellular to extracellular activities of 

Dynamic control of calcium, phosphate, citrate, and glucose levels in blood serum. Effect of ACTH, adrenaline, noradrenaline, hydrocortisone, parathormone, insulin, and glucagon.

Mode of Action of Insulin, Carbutamide, and Tolbutamide.∗

No qualitative differences were noted between the 2 routes of administration: in all cases

 caused a decrease in concentration of blood glucose and 

 and an increase in concentration of pyruvate and lactate.

Mechanisms of insulin and epinephrine effect on the level of plasma potassium.

 content of plasma, liver, and muscle are altered following the administration of 

 or epinephrine in the rat (Dury, 1951; Dury and Johnston, 1951; Dury and Moss, 1952).

The results of these investigations indicated that

 level in consequence of its effect upon carbohydrate assimilation and utilization when carbohydrate reserves are adequate or glucose is made available by exogenous supply (Dury, 1951; Dury and Moss, 1952; ...

The effect of epinephrine and insulin on the plasma potassium level.

 induced a rapid and significant fall in plasma 

 level in man and animals has been reported by several investigators (Houssay et al., 1937; D'Silva, 1937; Flocks et al., 1939; Keys, 1938a, 1938b; ...

Electrolyte balance in uncontrolled and controlled diabetic ketosis and acidosis.

Soon after the discovery of insulin, Harrop and Benedict (1), and shortly thereafter Briggs and co-workers (2), demonstrated a fall in the

Consequences of cerebroventricular insulin injection on renal sodium handling in rats: effect of inhibition of central nitric oxide synthase.

 produced an increase in urinary output of sodium (NaCl: 855.6 +/- 85.1 Delta%/min; 126 ng insulin: 2055 +/- 310.6 Delta%/min; P = 0.005) and 

 (NaCl: 460.4 +/- 100 Delta%/min; 126 ng insulin: 669.2 +/- 60.8 Delta%/min; P = 0.025).

Acute intracerebroventricular insulin microinjection after nitric oxide synthase inhibition of renal sodium handling in rats.

 produced increase in the urinary output of sodium (from 0.15 M NaCl: 855.…%.min(-1) to 126 ng insulin: 2055 +/- 310.6 Delta%.min(-1)) and 

 (126 ng: from 0.15 M NaCl: 460.4 +/- 100 … 126 ng insulin: 669 +/- 60.8 Delta%.min(-1)).

Effect of intracerebroventricularly injected insulin on urinary sodium excretion by cerebroventricular streptozotocin-treated rats.

 (42.0 ng/ micro l) increased the urinary output of sodium (from 855.6 85.1 to 2055 310.6 delta%/min; N = 11) and 

 (from 460.4 100 to 669 60.8 delta%/min; N = 11).

Betacellulin and activin A coordinately convert amylase-secreting pancreatic AR42J cells into insulin-secreting cells.

, tolbutamide, carbachol, and glucagon-like peptide-1 stimulated the release of immunoreactive 

Calcium and a mitochondrial signal interact to stimulate phosphoinositide hydrolysis and insulin secretion in rat islets.

 (30 mM), which depolarizes the membrane and increases Ca2+ influx, caused only a transient monophasic release of 

[Epinephrine decreases plasma potassium].

 (0.064 U.kg-1.h-1) combined with glucose (0.32 g.kg-1.h-1) or epinephrine (0.05, 0.1, 0.5, 1, or 5 micrograms.kg-1.min-1) added to 

 infusion significantly depressed the increments of plasma potassium levels.

Modulation of transmembrane potential of isolated cardiac myocytes by insulin and isoproterenol.

In conclusion, 1) hormonal modulation of cardiac myocyte membrane potentials … hyperpolarization by insulin and depolarization by beta-agonists, 2)

 conductance and may be antagonized by beta-stimulation, and 3) membrane potential modulation may be profoundly altered in the diabetic state.

Influence of insulin on plasma concentration and renal excretion of sodium and potassium in normal, electrolytes depleted and aldosterone treated dogs.

 in this Series elicited smaller decrease in plasma 

 concentration and longer lasting hypernatremia than in dogs in water-electrolytes balance.

Secretagogues for pancreatic hormone release in the channel catfish (Ictalurus punctatus).

 (25 mEq/liter) induced a pronounced release of 

 and glucagon and a moderate release of somatostatin.

Five new insulin-producing cell lines with differing secretory properties.

 (40 mmol/l) caused a three- to tenfold calcium-dependent increase in release of 

Perifusion of a Clonal Cell Line of Simian Virus 40-Transformed Beta Cells: Insulin Secretory Dynamics in Response to Glucose, 3-Isobutyl-1-Methylxanthine, and Potassium

 (40 mM) also triggered a monophasic release of 

The effect of potassium, calcium and magnesium concentration on insulin and glucagon secretion of the perfused dog pancreas.

 concentration above 15 mMol/l shortly stimulates the 

Glucose transfer studies in isolated cardiocytes of adult rats.

Plasma parathyroid hormone and insulin concentrations in cows administered potassium chloride and sodium citrate.

These results provide evidence for a relationship between

 and the endocrine factors, parathyroid hormone, and 

; citrate appears to antagonize tubular resorption of magnesium and calcium.

Blood plasma magnesium, potassium, glucose, and immunoreactive insulin changes in cows moved abruptly from barn feeding to early spring pasture.

 (2 IU/kg body weight) reduced plasma concentrations of both 

 and magnesium 20% below that of two cows injected with only physiological saline.

Insulin stimulation of (Na+,K+)-adenosine triphosphatase-dependent 86Rb+ uptake in rat adipocytes.

 stimulated the uptake of 86Rb+ (a K+ analog) in rat adipocytes and increased the steady state concentration of intracellular 

[Renal potassium excretion in rats exposed to different carbohydrate metabolism treatments].

 per 100 g of body weight along with administration of standard amount of 

 led to a decrease in the potassium excretion to 358.8 +/- 16.8 mueq/100 g.4 hours.

The effect of potassium and valinomycin on insulin and glucagon secretion in the perfused rat pancreas.

 caused a dose-response increase in both phases of 

, but did not increase glucagon secretion.

 from a baseline concentration of 5.8 mM to … 53.3 mM caused a biphasic release of immunoreactive 

 characterized by a spiked first phase and a low sustained second phase and a spiked … by a very low sustained secretion that ended with the termination of the stimulation.

Regulation of in vitro insulin release from a transplantable Syrian hamster insulinoma.

 release was increased 2-4 fold by 40 mM 

 in the presence of calcium, glucose (22 mM), glucagon (0.3-3.0 muM), N6,02'-dibutyryl adenosine 3',5'-monophosphate (cAMP; 6mM), and theophylline (10 mM).

Possible mechanisms of insulin action on membrane potential and ion fluxes.

 that one can infuse insulin into the brachial artery of man at a rate that elevates brachial arterial insulin concentration by only 30 punits per ml.

Separation of insulin effects on K content and O2 consumption of frog muscle with cardiac glycosides.

 and ouabain together caused a greater net loss of 

Efficacy of standard- vs reduced-dose insulin for treatment of hyperkalemia: A quasi-experiment

 intracellularly may cause hypoglycemia, requiring additional treatment or longer hospitalization.

Acute hyperglycemic emergencies in children with type 2 diabetes.

In transferring these children from remote communities to paediatric centres for intensive medical care, intravenous administration … at no more than twice the normal fluid maintenance rate is vital; intravenous (or subcutaneous)

 are not necessary in transit because there is … for either therapy, and both are potentially dangerous.

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Possible Interaction: Insulin and Potassium

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Research Papers that Mention the Interaction