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“Additionally, apigenin was able to induce mitochondrial redox impairment, once it increased ROS levels and H2O2 , decreased the Δψm, and increased LPO.”
Oxidative medicine and cellular longevity • 2017 | View Paper
“Conclusion The … suggest that apigenin may protect … embryos against H2O2 and actinomycin D. So that it increases the number of viable blastomeres and decreases the number of apoptotic blastomeres, which may cause expanding the blastocysts, thinning of the ZP thickness and increasing the rate of hatching in mouse embryos.”