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“Administration of the nitric oxide (NO) synthase blocker, N(G)-monomethyl L-arginine prevents the increase in glomerular filtration rate (GFR) normally observed with glycine , an effect that is restored by angiotensin II (AII) blockers.”
American journal of kidney diseases : the official journal of the National Kidney Foundation • 1996 | View Paper
“The NO synthase blocker, NG-monomethyl L-arginine (LNMMA), abolished the vasodilatory response to glycine , possibly through activation of tubuloglomerular feedback due to a decrease in APR produced by LNMMA + glycine.”