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Last Updated: 3 years ago

Possible Interaction: Glycerol and Propranolol

supplement:

Glycerol

Research Papers that Mention the Interaction

Only propranolol produced a significant decrease (P < .05) in the rate of appearance of glycerol , from a mean (+/- SD) of 5.54 +/- 0.62 to 3.07 +/- 0.7 mumol/kg per minute.
Archives of surgery  •  1994  |  View Paper
Propranolol reduced fasting glycerol and free fatty acid concentrations in both groups.
The American journal of clinical nutrition  •  1992  |  View Paper
Propranolol decreased the pulse rate (P less than 0.01) and the levels of serum triiodothyronine (T3; P less than 0.05), blood glycerol (P less than 0.01), and KB (P less than 0.01).
Metabolism: clinical and experimental  •  1984  |  View Paper
Apart from possible effects on hepatic glycogenolysis propranolol also inhibits the release of the important gluconeogenic substrates lactate and glycerol.
Clinical science  •  1982  |  View Paper
Propranolol has been shown also to perturb glycerol transport in red cells.
Biochemical pharmacology  •  1981  |  View Paper
Despite an identical exercise time, glycerol and NEFA concentrations during recovery were significantly lower after propranol treatment.
European Journal of Clinical Pharmacology  •  2004  |  View Paper
In hyperthyroid patients propranolol administration for 4 days induced a decrease in triiodothyronine (349±140 to 229±107 ng/100ml, p<0.01) and a dramatic fall in ketone body (18–295 μmol/l, p<0.001) and glycerol (44–130 μmol/l, p<0.001) levels.
Diabetologia  •  2004  |  View Paper
A corresponding local beta-adrenoceptor blockade with propranolol (10−4 M), inhibited the increase in adipose glycerol levels by ∼60% (P=0.02).
International Journal of Obesity  •  2001  |  View Paper
The mechanism of the effect of propranolol is probably an attenuation of the gluconeogenesis secondary to deficient release of the important gluconeogenic substrates lactate and glycerol.
Acta medica Scandinavica. Supplementum  •  1983  |  View Paper
Oral propranolol administration to hyperthyroid patients in the post-absorptive state decreased the concentrations of glycerol (p less than 0.05) and ketone bodies (p less than 0.05) without altering insulin concentrations.
Diabete & metabolisme  •  1983  |  View Paper
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