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World journal of gastroenterology • 2018 | View Paper
“ConclusionThus, changes in GSH levels with exogenous agents such as NAC, UDCA , BSO or DEM modulate hyperglycemia-mediated injury in a cell model of VL-17A liver cells.”
“In conclusion, modulation of GSH with NAC or UDCA or BSO or DEM leads to significant changes in oxidative stress, glyoxalase 1 enzyme activity and toxicity in chronic alcohol plus high glucose treated VL-17A cells.”
“In conclusion, UDCA treatment enhances the antioxidant defense mediated by glutathione ; in doing so, this treatment prevents cardiolipin depletion and cell injury in animals with secondary biliary cirrhosis. (”
“Results: We found that in livers perfused with ursodeoxycholate , levels of glutathione and the activity of methionine adenosyltransferase (an enzyme involved in glutathione biosynthesis) were significantly higher than in livers perfused with other bile salts.”
“In conclusion, UDCA increased hepatocyte levels of GSH and thiol-containing proteins such as MT, thereby protecting hepatocytes against oxidative injury.”
Biochemical and biophysical research communications • 1999 | View Paper
“Reduced glutathione GSH ) levels tended to slightly decrease with UDCA diets but the difference did not attain a statistical significance (P > 0.05, one-way ANOVA).”