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Last Updated: 3 years ago

Possible Interaction: Gamma-Aminobutyric Acid and Potassium

supplement:

Potassium

Research Papers that Mention the Interaction

Based on this information, it was found that GABA also activated a potassium conductance.
Biochemical pharmacology  •  2004  |  View Paper
High potassium pulses stimulate release of, for example GABA in both neuronal and glial cells.
Journal of neuroscience research  •  1975  |  View Paper
Perfusion with a high concentration of potassium (100 mM) increased GABA , but not glutamate, release and thioperamide (20 mg/kg) attenuated the effects of high potassium on GABA release.
Psychopharmacology  •  2009  |  View Paper
High potassium and electrical stimulation consistently increase efflux of labelled GABA from the in vivo superfused rat dorsal medulla in a calcium-dependent fashion.
Experientia  •  2005  |  View Paper
In the synaptosomal fraction, 68.5 mM potassium significantly stimulated the efflux of GABA and glycine by a calcium-dependent mechanism.
Neurochemical Research  •  2004  |  View Paper
SummaryGamma-aminobutyric acid depressed the potassium contraction of the skeletal muscles and its sensitization to potassium ions caused by guanidine, aminopyridine, veratrine, and had no material effect on the aceytlcholine muscle contractions.
Bulletin of Experimental Biology and Medicine  •  2004  |  View Paper
Although high potassium evoked the release of both excitatory (glutamate and aspartate) and inhibitory ( GABA and glycine) amino acid transmitters, BDNF only induced glutamate and aspartate release.
Neuroscience Research  •  2000  |  View Paper
Potassium evoked the Ca2+‐dependent release of [3H]GABA and [3H]acetylcholine, with EC50 of 31.0 ± 1.0 mm and 21.6 ± 1.1 mm, respectively.
The European journal of neuroscience  •  1998  |  View Paper
4 GVG and EOS administration led to a significant elevation in the potassium stimulated release of GABA from cross‐chopped hippocampal slices compared with that of controls.
British journal of pharmacology  •  1997  |  View Paper
Application of GABA (50 microM) reduced the amplitude of A-current when potassium current amplitude was measured during a period of slow and incomplete desensitization of IGABA.
Journal of neurophysiology  •  1996  |  View Paper
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