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Discover Supplement-Drug Interactions
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Last Updated: 3 years ago
supplement:
Cupric Cation
Research Papers that Mention the Interaction
“Because Cu2+ chelation also represses the release of FGF1 , the ability of Cu2+ chelators to potentially serve as effective clinical anti-cancer agents may be related to their ability to limit the export of these proinflammatory and angiogenic signal peptide-less polypeptides into the extracellular compartment.”
S100A13 mediates the copper-dependent stress-induced release of IL-1α from both human U937 and murine NIH 3T3 cells
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