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Possible Interaction: Ethanol and Hydrogen Peroxide



Research Papers that Mention the Interaction

In parallel, acute ethanol in the presence of H2O2 , but neither ethanol nor H2O2 alone, stimulated transient receptor potential canonical 6 (TRPC6) channels and caused TRPC6-dependent elevation of intracellular Ca2+.
Biochimica et biophysica acta  •  2015  |  View Paper
In this study, we investigated the possible protective effects of ethanol and aqueous extracts of rosemary on human lymphocyte DNA damage induced by H2O2.
Journal of complementary & integrative medicine  •  2014  |  View Paper
Ethanol (50 mM) induced H2O2 production in platelets by Ca2+-dependent and independent mechanisms.
In conclusion we provide considerable evidence that ethanol alters Ca2+ entry and reduces thrombin-induced aggregation as a result of the generation of H2O2 and the oxidation of sulphydryl groups in human platelets.
Archives of biochemistry and biophysics  •  2006  |  View Paper
Combinations other than AE + H2O2 showed possible interactions, which lead to reduction in their potency.
Biomedical sciences instrumentation  •  2003  |  View Paper
The results obtained showed that ethanol , glycerol, the catechin-caffeic acid mixture, the mixture of all components and the stripped white wine significantly reduced the DNA-damaging effects of hydrogen peroxide and gamma-radiation (P = 0.043-0.001, ANOVA).
These two treatments as well as ethanol produced the strongest protective effects against DNA damage by hydrogen peroxide (24, 25 and 18%, respectively).
Mutagenesis  •  2003  |  View Paper
as a consequence of ethanol drinking and resultant endogenous synthesis of 8,9A, and 9B would, based on these in vitro chemical studies, be expected to generate elevated fluxes of H2O2 and HO.
Journal of medicinal chemistry  •  1996  |  View Paper
Addition of 0.4% ethanol had no effect on superoxide concentration (102.2%), but tended to decrease hydrogen peroxide (83.5%) and 8OHdG (84.3%).
Carcinogenesis  •  1996  |  View Paper
Similarly, ethanol treatment completely blocked H2O2 and lactoferrin release, and the second phase of DRG formation.
The Journal of biological chemistry  •  1994  |  View Paper
The generation of aberrations by H2O2 was effectively suppressed by catalase and several scavengers of hydroxyl radicals (.OH) such as ethanol , dimethyl sulfoxide (DMSO) and mannitol.
Mutation research  •  1986  |  View Paper
The results showed that ethanol stress very significantly induced expression of ATG1 and ATG8 genes and the production of peroxide hydrogen (H₂O₂) and superoxide anion (O₂·-).
Journal of microbiology and biotechnology  •  2018  |  View Paper
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