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Last Updated: 2 years ago

Possible Interaction: Ethanol and Aspartic Acid

supplement:

Ethanol

supplement:

Aspartic Acid

Research Papers that Mention the Interaction

In silico modeling suggested that ethanol alters the dynamics for assembling Aβ by disrupting a critical salt bridge between residues Asp 23 and Lys 28, required for amyloid dimerization.
Archives of medical research  •  2013  |  View Paper
Those living in states with a MADD grade of D ” were 60% more likely to report alcohol impaired driving than those from states with a MADD grade of “A” (odds ratio 1.6, 95% confidence interval 1.3 to 2.1).
Injury prevention : journal of the International Society for Child and Adolescent Injury Prevention  •  2002  |  View Paper
An acute intoxication result in transitory hypoparatthyreoidism, while chronic ethanol intake make grow the PTH level and decreases the level of D vitamin metabolises.
Sbornik lekarsky  •  1993  |  View Paper
The results clearly indicated that ethanol promoted the release of both Asp and Glu from the cells.
Analytical and Bioanalytical Chemistry  •  2013  |  View Paper
In contrast, several kinases that participate in the metabolism of these phospholipids, and the level and/or activity of phospholipases C and D , increase in cells after ethanol exposure.
Alcohol and alcoholism  •  2013  |  View Paper
NMDA activation during EtOH exposure is necessary for withdrawal hyper‐responsiveness.
When NMDA applications were stopped durithe period of EtOH exposure, mean area of NMDA‐evoked responses on washout was only 98.0±5% of control (n=6, P>0.05).
British journal of pharmacology  •  2003  |  View Paper
Ethanol lowered the concentration of most plasma amino acids (AA) or related compounds in stressed rats (e.g., aspartic acid , threonine, serine, glycine, alanine, valine, tyrosine, phenylalanine, tryptophan).
Pharmacology Biochemistry and Behavior  •  1989  |  View Paper
The rate of sRNA aminoacylation by aspartic acid was increased in presence of 85 mmole of ethanol.
Voprosy meditsinskoi khimii  •  1988  |  View Paper
Chronic ethanol treatment produced a decrease in the arteriovenous differences of Asp , Thr, Arg, Met and Phe, and increased those of Ala, Gln, Gly, Pro and Tyr.
Ethanol treatment decreased the arterial levels of Ala, Asp , Gly, Pro, Lys and Met, and increased those of Gln and alpha-amino-butyrate.
Life sciences  •  1987  |  View Paper
Addition of aspartic acid , which can restore Krebs cycle intermediates, partially restored growth on ethanol.
Biochimica et biophysica acta  •  2006  |  View Paper
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