Allen Institute for Artificial Intelligence
supp.ai logo
supp.ai

Discover Supplement-Drug Interactions

Disclaimer: The information contained herein should NOT be used as a substitute for the advice of an appropriately qualified and licensed physician or other health care provider. The tool is not a substitute for the care provided… (more)
Last Updated: 2 months ago

Possible Interaction: Epinephrine and Tretinoin

supplement:

Epinephrine

supplement:

Tretinoin

Research Papers that Mention the Interaction

Aim of this study was to evaluate whether retinoic acid modulates cortisol synthesis and secretion by human adrenals in vitro.
Life sciences  •  2016  |  View Paper
In any of these basal media, addition of 1,25-dihydroxyvitamin D(3), all-trans-retinoic acid or dexamethasone significantly increased activity of the nephrin promoter.
American journal of physiology. Renal physiology  •  2007  |  View Paper
After its autophosphorylation by ecto-kinase activity, a 98-kDa membrane protein in … sensitive to ecto-ALP, and protein dephosphorylation increased after incubation of cells with RA for 24 h and 48 h. Orthovanadate, an inhibitor … levamisole-sensitive dephosphorylation of the membrane phosphoproteins, while (R)-(-)-epinephrine reversed the … inhibitor.
Journal of cellular biochemistry  •  2000  |  View Paper
Compared with the injured podocytes, ATRA exposure significantly increased the protein/mRNA expression of nephrin and podocin and it markedly reduced TGF-β1 (all p < 0.05).
Journal of receptor and signal transduction research  •  2014  |  View Paper
ATRA also significantly prevented the decrease in staining for synaptopodin, nephrin , and podocin in experimental animals (P < 0.05 vs. control).
Kidney international  •  2005  |  View Paper
The effect of retinoic acid on cardiac output could not be compensated by epinephrine application.
The effect of retinoic acid on cardiac output could not be compensated by epinephrine application.
Fetal Diagnosis and Therapy  •  2004  |  View Paper
In SupB- cells, transforming growth factor-beta 1 and retinoic acid inhibited anchorage-independent growth response to EPI but not the growth response to bFGF.
Molecular carcinogenesis  •  1993  |  View Paper