Allen Institute for Artificial Intelligence
supp.ai logo
supp.ai

Discover Supplement-Drug Interactions

Disclaimer: The information contained herein should NOT be used as a substitute for the advice of an appropriately qualified and licensed physician or other health care provider. The tool is not a substitute for the care provided… (more)
Last Updated: a month ago

Possible Interaction: Epinephrine and Adrenergic Antagonists

Research Papers that Mention the Interaction

In contrast, when plasma glucose, insulin, and glucagon were clamped, beta adrenergic blockade completely prevented the suppression of glucose clearance by epinephrine and inhibited the stimulation of glucose production by epinephrine by 80% whereas alpha adrenergic blockade had no effect on either of these parameters.
Metabolism: clinical and experimental  •  1980  |  View Paper
The adrenergic blocking agents prevent the pressor responses to the circulating epinephrine and nor-epinephrine liberated from the adrenal tumors.
The American journal of medicine  •  1951  |  View Paper
A large dose of epinephrine can be both tapenolytic and adrenolytic.
The Journal of pharmacology and experimental therapeutics  •  1949  |  View Paper
In addition substances other than adrenergic blocking agents (e.g., posterior pituitary) are able to inhibit the hyperglycemic response to epinephrine.
The Journal of pharmacology and experimental therapeutics  •  1949  |  View Paper
Properly an adrenolytic agent is one which nullifies an important effect of epinephrine , whereas a sympatholytic agent is one which nullifies sympathetic nerve transmission, usual reference being to adrenergic pathways controlling blood vessels and usual site of action being at the end organ.
Journal of the American Medical Association  •  1948  |  View Paper
It is reported to produce peripheral vasodilatation by: 1) adrenergic blockade , inhibiting the effects of circulating adrenalin on smooth muscle cells of blood vessels; 2) sympathetic blockade at the terminations of the sympathetic nerves in the blood vessels; 3) direct action on peripheral vessels; and 4) epinephrine-reversal.
The Journal of clinical investigation  •  1953  |  View Paper
In conclusion, portal delivery of adrenergic blockers selectively inhibits the glycogenolytic effects of EPI and NE on the liver, but allows a marked gluconeogenic response to the catecholamines.
Metabolism: clinical and experimental  •  1997  |  View Paper
The larval receptors mediating this induction were pharmacologically characterized by testing the ability of a variety of adrenergic agonists and selected structural analogs of epinephrine and norepinephrine to induce oyster metamorphosis, and by testing the ability of various adrenergic antagonists to block the induction of metamorphosis by epinephrine.
Neuroscience  •  1987  |  View Paper
Epinephrine (3 X 10(-7) M)-stimulated PGI2 synthesis was inhibited by adrenoreceptor antagonists (rank order of potency: yohimbine greater than prazosin greater than phentolamine greater than corynanthine much greater than propranolol).
European journal of pharmacology  •  1985  |  View Paper
Adrenergic blocking agents also inhibited the suppressive effect of epinephrine.
Journal of the autonomic nervous system  •  1980  |  View Paper
Show More