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Possible Interaction: Dehydroepiandrosterone Sulfate and Adrenocorticotropic Hormone

Research Papers that Mention the Interaction

In addition, the correlation was highly significant (P<0.001) between ACTH and DHEA-S at the term of HRT.
Maturitas  •  2001  |  View Paper
ACTH administration caused a definite increase in plasma DS as compared to the control values, and there was no difference between young and aged groups in per cent increase in plasma DS values after ACTH stimulation.
The Journal of clinical endocrinology and metabolism  •  1969  |  View Paper
ACTH treatment enhanced the secretion of cortisol and to a lesser extent that of DHAS , and increased the steroidogenic response to an acute stimulation with ACTH.
Molecular and Cellular Endocrinology  •  1994  |  View Paper
In this homosexual man, however, the plasma dehydroepiandrosterone sulfate (DHEA-S) values and the DHEA-S/1000 x A (A = androstenedione) ratio were increased before and after ACTH stimulation.
Experimental and clinical endocrinology  •  1991  |  View Paper
Continuous treatment with 1 nM ACTH increased dehydroepiandrosterone sulfate (DHAS) production 5- to 10-fold during the first 5 days in culture.
Biology of reproduction  •  1990  |  View Paper
The addition of adrenocorticotropic hormone , forskolin, 22R-hydroxycholesterol, or dibutyryl cyclic adenosine monophosphate increased the secretion of dehydroepiandrosterone sulfate and cortisol twofold to threefold and twofold to sixfold, respectively, above basal rates.
When cells were incubated in the presence of adrenocorticotropic hormone , forskolin, 22R-hydroxycholesterol, or dibutyryl cyclic adenosine monophosphate plus atrial natriuretic polypeptide, cortisol secretion was inhibited 50% to 90% and dehydroepiandrosterone sulfate was inhibited 25% to 50%.
American journal of obstetrics and gynecology  •  1988  |  View Paper
DS secretion increased up to 5-fold and pregnenolone about 2-fold in the presence of ACTH compared to values in control cells.
Endocrinology  •  1987  |  View Paper
ACTH caused significant increases in the productions of pregnenolone (P5), progesterone (P4), 17-hydroxypregnenolone (17-OH-P5), 17-hydroxyprogesterone (17-OH-P4), DHEA, DHEA-S , androstenedione (delta 4-A), and cortisol.
Endocrinologia japonica  •  1985  |  View Paper
In groups II and III mean basal levels of plasma dehydroepiandrosterone sulphate were also significantly increased and showed a marked increase after ACTH stimulation (1 mg tetracosactide acetate, i.m.)
The Journal of endocrinology  •  1983  |  View Paper
It is considered that the responsiveness of DHEA-S to ACTH may increase in the low and normal PRA groups and that the responsiveness of DHEA-S to angiotensin II may increase in the high PRA group.
Japanese circulation journal  •  1982  |  View Paper
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