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Last Updated: a year ago

Possible Interaction: Carbachol and Tubocurarine

supplement:

Tubocurarine

Research Papers that Mention the Interaction

Moreover, it shows that carbachol exerts an additional agonistic action on nicotinic cholinergic receptor of prostate cancer cells that can be blocked by tubocurarine.
Anti-cancer drugs  •  2017  |  View Paper
In contrast, 1--10 nM carbamoylcholine produced diminished in vitro proliferation, an effect which was blocked by the nicotinic antagonists alpha-bungarotoxin and d-tubocurarine.
Proceedings of the National Academy of Sciences of the United States of America  •  1979  |  View Paper
Application of a somewhat larger quantity of DTC (about 4 x 10-10 C) produces a transient 50 % inhibition of the acetylcholine (or carbachol ) potential.
Proceedings of the Royal Society of London. Series B - Biological Sciences  •  1957  |  View Paper
The effect of carbachol was blocked by two nicotinic blockers, mecamylamine (10−6 M) and d-tubocurarine (10−6 M).
Naunyn-Schmiedeberg's Archives of Pharmacology  •  2004  |  View Paper
2) Carbachol increased the uptake of 86Rb+; this increase was inhibited by hexamethonium and d-tubocurarine.
Naunyn-Schmiedeberg's Archives of Pharmacology  •  2004  |  View Paper
D-tubocurarine reduced the effect of carbamylcholine.
The paired-pulse facilitation of the synaptic potentials changed significantly under the action of carbamylcholine and d-tubocurarine.
Visual Neuroscience  •  2001  |  View Paper
Carbachol , a non-specific cholinergic agonist, selectively induced phosphorylation of TH at serine 31 in darkness, an effect which was blocked by the nicotinic antagonist, d-tubocurarine.
Journal of Chemical Neuroanatomy  •  2000  |  View Paper
The iontophoretic carbachol response is antagonized by alpha-bungarotoxin (EC50 = 0.19 microM), mecamylamine (EC50 = 0.32 microM), d-tubocurarine (EC50 = 9.5 microM), and bicuculline but not by decamethonium and scopolamine.
Journal of neurophysiology  •  1996  |  View Paper
The uncoupling effects of carbachol and DMPP were blocked by the nicotinic antagonist D-tubocurarine and by the dopamine antagonist fluphenazine, indicating that carbachol uncoupled horizontal cells by stimulating dopamine release via nicotinic receptors.
Vision Research  •  1996  |  View Paper
Tubocurarine produced a concentration-dependent reduction in the EMG responses, EPP amplitude, mean quantal content and carbachol sensitivity.
British journal of anaesthesia  •  1995  |  View Paper
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