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“This mechanism could also be active in vivo as we have observed that cytosolic type‐II EBS are present in primary colorectal cancers and that Q is effective in inhibiting the in vitro bromodeoxyuridine incorporated by neoplastic cells in these cancers.”
International journal of cancer • 1992 | View Paper
“Furthermore, Gln increased the cellular population of both Oct4 and bromodeoxyuridine positive cells , suggesting that Gln regulates self-renewal ability of mESCs.”