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Last Updated: 4 months ago

Possible Interaction: Bismuth Subsalicylate and Kaempferol

Research Papers that Mention the Interaction

ConclusionsOur results suggest that luteolin induces apoptosis through activating ER stress and mitochondrial dysfunction in glioblastoma cell lines and in vivo, which provides the anti-cancer candidate to treat glioblstoma.
Cancer Chemotherapy and Pharmacology  •  2017  |  View Paper
Indeed, the inhibition of ER stress suppressed kaempferol-induced autophagy and conferred prolonged cell survival, indicating autophagic cell death.
Cell Death & Disease  •  2018  |  View Paper
Altogether, we have demonstrated the ability of kaempferol to alleviate ER stress in in vitro model.
We have shown that kaempferol pre-incubation significantly inhibits the expression of GRP78 (a chaperone) and CHOP ( ER stress associated pro-apoptotic transcription factor) under stressed condition.
Scientific Reports  •  2018  |  View Paper
Furthermore, luteolin increased the level of intracellular ROS, leading to ROS-mediated apoptosis and ER stress.
Anticancer research  •  2016  |  View Paper
Together, these findings suggest that kaempferol prevents ectopic lipid accumulation and ER stress , thus restoring β-cell function through AMPK-mediated lipophagy.
The Journal of nutritional biochemistry  •  2018  |  View Paper
In conclusion, luteolin improved renal function in I/R rats by reducing oxidative stress, neutrophil infiltration, inflammation, renal cell apoptosis, and expression of HMGB1 and NF-κB, and ER stress.
Mediators of inflammation  •  2017  |  View Paper
In ex vivo-Langendorff experiment, the kaempferol treatment regulated the expression of ER stress proteins-CHOP and GRP78.
Immunopharmacology and immunotoxicology  •  2008  |  View Paper
Our results demonstrated that kaempferol induced hepatocarcinoma cell death via ER stress and CHOP-autophagy signaling pathway; kaempferol may be used as a potential chemopreventive agent for patients with hepatocellular carcinoma.
Oncotarget  •  2017  |  View Paper
Our results demonstrated that kaempferol induced hepatocarcinoma cell death via ER stress and CHOP-autophagy signaling pathway; kaempferol may be used as a potential chemopreventive agent for patients with hepatocellular carcinoma.
Oncotarget  •  2017  |  View Paper