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“Therefore, it is important to examine whether fenofibrate would modulate the cardiac P450 and its associated arachidonic acid metabolites and whether this modulation protects against isoproterenol-induced cardiac hypertrophy.”
Journal of cardiovascular pharmacology • 2014 | View Paper
“Release of prostaglandin (PG) E2 in response to AA was enhanced in the diabetic rat kidney compared with control (4.8 ± 0.7 versus 1.9 ± 0.7 ng/min) and reduced by fenofibrate to 0.6 ± 0.2 ng/min.”