Disclaimer: The information contained herein should NOT be used as a substitute for the advice of an appropriately qualified and licensed physician or other health care provider. The tool is not a substitute for the care provided… (more)
“The synergistic effect was blocked by the α2-adrenoceptor antagonist yohimbine and inhibited by the ADP scavenger system creatine phosphate/creatine phosphokinase and antibodies against αIIbβ3.”
Blood coagulation & fibrinolysis : an international journal in haemostasis and thrombosis • 2006 | View Paper
“The adrenergic blocking agents, Dibenamine, yohimbine , benzodioxanes, and Pris-coline, when given in divided doses, progressively block epinephrine and nor-epinephrine pressor effects as the doses of the blocking agents are increased.”
Journal of the American Pharmaceutical Association. American Pharmaceutical Association • 1951 | View Paper
“We previously revealed that pre-treatment with yohimbine or JP-1302 attenuated renal ischemia/reperfusion injury by inhibition of α2C-adrenoceptor antagonist.”
Journal of pharmacological sciences • 2019 | View Paper
“Furthermore, loser effects were partly eliminated by either injection of epinastine, an octopamine blocker, or yohimbine , a tyramine blocker, and significantly diminished by injection of a mixture of both blockers.”
Journal of Neural Transmission • 2005 | View Paper
“Mice implanted for 5 days with osmotic minipumps containing the α2-adrenoceptor agonist clonidine showed symptoms of a withdrawal syndrome (jerks, headshakes, defecations and weight loss) when yohimbine , an α2-adrenoceptor antagonist, was injected.”
Naunyn-Schmiedeberg's Archives of Pharmacology • 2004 | View Paper
“During stimulation with 36 pulses at 3 Hz the α2-adrenoceptor antagonist yohimbine induced a five-fold increase of noradrenaline release indicating a pronounced autoinhibition of approximately 80%.”
Naunyn-Schmiedeberg's Archives of Pharmacology • 2004 | View Paper
“Pretreatment with either the beta-adrenoceptor antagonist nadolol or the selective alpha2-adrenoceptor antagonist yohimbine prevented both leukocyte peaks in the high volume group, suggesting a combined receptor activation.”
“Furthermore, these alpha 2- and beta-agonistic effects were almost completely inhibited in the presence of the alpha 2-adrenoceptor antagonist yohimbine and the beta-adrenoceptor antagonist propranolol at a concentration of 10(-6) mol/L, respectively.”