“In isolated hearts, midazolam (5, 10, 20 &mgr;M) significantly augmented coronary flow in a concentration-dependent manner in the presence of adenosine (30 nM), an effect reversed by ZM 241385, a selective A2A-receptor antagonist.”
“ Midazolam selectively augments A2A- but not A1-receptor–mediated effects of adenosine by inhibiting nucleoside transporter.”