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“Initiating oral GlcNAc treatment on the second day of clinical disease inhibited MOG-induced EAE as well as secretion of interferon-γ , tumor necrosis factor-α, interleukin-17, and interleukin-22.”
The Journal of Biological Chemistry • 2011 | View Paper
“The stimulation of (pro)insulin biosynthesis by adenosine, D-glyceraldehyde, mannose, N-acetylglucosamine and leucine was also inhibited by interferon , with no effect on insulin release.”